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active rac1 detection kit  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc active rac1 detection kit
    Active Rac1 Detection Kit, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 95/100, based on 167 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/active rac1 detection kit/product/Cell Signaling Technology Inc
    Average 95 stars, based on 167 article reviews
    active rac1 detection kit - by Bioz Stars, 2026-03
    95/100 stars

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    rac1 activity  (Cell Signaling Technology Inc)
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    Cell Signaling Technology Inc rac1 activity
    Differential gene enrichment analysis of renal endothelial cells from IgAN and control kidney tissues. (A) Cell adhesion was enriched in the BP of GO analysis. (B) Cell‐substrate junctions and focal adhesions were enriched in the CC of GO analysis. (C) Cadherin binding was further targeted in the MF analysis of GO analysis. (D) KEGG analysis showed enrichment of the focal adhesion pathway. (E) The hub genes HSP90AA1, <t>RAC1,</t> EEF1A1, FN1, GAPDH, PPARA, EHHADH, ACTB, HSP90AB1, and CAV1 were identified through CytoHubba analysis using the bottleneck method. BP, biological process; CC, cellular component; GO, gene ontogeny; IgAN, IgA nephropathy; KEGG, Kyoto Encyclopedia of Genes and Genomes; MF, molecular function.
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    Differential gene enrichment analysis of renal endothelial cells from IgAN and control kidney tissues. (A) Cell adhesion was enriched in the BP of GO analysis. (B) Cell‐substrate junctions and focal adhesions were enriched in the CC of GO analysis. (C) Cadherin binding was further targeted in the MF analysis of GO analysis. (D) KEGG analysis showed enrichment of the focal adhesion pathway. (E) The hub genes HSP90AA1, RAC1, EEF1A1, FN1, GAPDH, PPARA, EHHADH, ACTB, HSP90AB1, and CAV1 were identified through CytoHubba analysis using the bottleneck method. BP, biological process; CC, cellular component; GO, gene ontogeny; IgAN, IgA nephropathy; KEGG, Kyoto Encyclopedia of Genes and Genomes; MF, molecular function.

    Journal: Immunity, Inflammation and Disease

    Article Title: Single‐Cell Analysis of Endothelial Cell Injury in IgA Nephropathy

    doi: 10.1002/iid3.70149

    Figure Lengend Snippet: Differential gene enrichment analysis of renal endothelial cells from IgAN and control kidney tissues. (A) Cell adhesion was enriched in the BP of GO analysis. (B) Cell‐substrate junctions and focal adhesions were enriched in the CC of GO analysis. (C) Cadherin binding was further targeted in the MF analysis of GO analysis. (D) KEGG analysis showed enrichment of the focal adhesion pathway. (E) The hub genes HSP90AA1, RAC1, EEF1A1, FN1, GAPDH, PPARA, EHHADH, ACTB, HSP90AB1, and CAV1 were identified through CytoHubba analysis using the bottleneck method. BP, biological process; CC, cellular component; GO, gene ontogeny; IgAN, IgA nephropathy; KEGG, Kyoto Encyclopedia of Genes and Genomes; MF, molecular function.

    Article Snippet: In this study, Rac1 activity was measured using a pull‐down assay kit (catalog no. 8815; Cell Signaling Technology, Danvers, MA, USA) as directed by the manufacturer.

    Techniques: Control, Binding Assay

    Effect of IL‐6 on VE‐cad expression in HRGECs. (A) IL‐6/IL‐6R had no effect on Rac1 expression in HRGECs, but increased Rac1‐GTP expression ( p < 0.01). The Rac1 inhibitor NSC23766 reduced the increase in Rac1‐GTP expression ( p < 0.01). (B) IL‐6/IL‐6R stimulation reduced the expression of VE‐cad in HRGECs ( p < 0.01); NSC23766 ameliorated this effect ( p < 0.01, n = 3). * p < 0.01, compared with the control; # p < 0.01, compared with the IL‐6/IL‐6R group; IL‐6, interleukin‐6; IL‐6R, interleukin‐6 receptor; HRGECs, human renal glomerular endothelial cells; VE‐cad, vascular endothelial cell cadherin.

    Journal: Immunity, Inflammation and Disease

    Article Title: Single‐Cell Analysis of Endothelial Cell Injury in IgA Nephropathy

    doi: 10.1002/iid3.70149

    Figure Lengend Snippet: Effect of IL‐6 on VE‐cad expression in HRGECs. (A) IL‐6/IL‐6R had no effect on Rac1 expression in HRGECs, but increased Rac1‐GTP expression ( p < 0.01). The Rac1 inhibitor NSC23766 reduced the increase in Rac1‐GTP expression ( p < 0.01). (B) IL‐6/IL‐6R stimulation reduced the expression of VE‐cad in HRGECs ( p < 0.01); NSC23766 ameliorated this effect ( p < 0.01, n = 3). * p < 0.01, compared with the control; # p < 0.01, compared with the IL‐6/IL‐6R group; IL‐6, interleukin‐6; IL‐6R, interleukin‐6 receptor; HRGECs, human renal glomerular endothelial cells; VE‐cad, vascular endothelial cell cadherin.

    Article Snippet: In this study, Rac1 activity was measured using a pull‐down assay kit (catalog no. 8815; Cell Signaling Technology, Danvers, MA, USA) as directed by the manufacturer.

    Techniques: Expressing, Control